Sula Karreci E, Jacas S, Donovan O, Pintye D, Wiley N, Zsengeller ZK, Schlondorff J, Alper SL, Friedman DJ, Pollak MR. Differing sensitivities to angiotensin converting enzyme inhibition of kidney disease mediated by APOL1 high-risk variants G1 and G2 , Kidney Int, 106(6): 2024,1072-1085
Madhavan SM, Schlöndorff JS. Variant upon variant: kidney-disease risk associated with APOL1 G2 genetic variants is abrogated by the APOL1 p.N264K variant , Kidney Int, 106(3): 2024,345-348
Thomas CP, Daloul R, Lentine KL, Gohh R, Anand PM, Rasouly HM, Sharfuddin AA, Schlondorff JS, Rodig NM, Freese ME, Garg N, Lee BK, Caliskan Y. Genetic evaluation of living kidney donor candidates: A review and recommendations for best practices , Am J Transplant, 23(5): 2023,597-607
Brown BJ, Boekell KL, Stotter BR, Talbot BE, Schlondorff JS. Gain-of-function, focal segmental glomerulosclerosis Trpc6 mutation minimally affects susceptibility to renal injury in several mouse models , PLoS One, 17: 2022,e0272313
Boekell KL, Brown BJ, Talbot BE, Schlondorff JS. Trpc6 gain-of-function disease mutation enhances phosphatidylserine exposure in murine platelets , PLoS One, 17: 2022,e0270431
Sun H, Perez-Gill C, Schlöndorff JS, Subramanian B, Pollak MR. Dysregulated Dynein-Mediated Trafficking of Nephrin Causes INF2-related Podocytopathy , J Am Soc Nephrol, 32(2): 2021,307-322
Schlöndorff JS. My, oh, MYO9A! Just how complex can regulation of the podocyte actin cytoskeleton get? , Kidney Int, 99(5): 2021,1065-1067
Pandit A, Schlondorff J. Posaconazole-Induced Apparent Mineralocorticoid Excess , Kidney Int Rep, 5(12): 2020,2379-2382
Feng D, Kumar M, Muntel J, Gurley SB, Birrane G, Stillman IE, Ding L, Wang M, Ahmed S, Schlondorff J, Alper SL, Ferrante T, Marquez SL, Ng CF, Novak R, Ingber DE, Steen H, Pollak MR. Phosphorylation of ACTN4 Leads to Podocyte Vulnerability and Proteinuric Glomerulosclerosis , J Am Soc Nephrol, 31(7): 2020,1479-1495
Stotter BR, Talbot BE, Capen DE, Artelt N, Zeng J, Matsumoto Y, Endlich N, Cummings RD, Schlondorff JS. Cosmc-dependent mucin-type O-linked glycosylation is essential for podocyte function , Am J Physiol Renal Physiol, 318(2): 2020,F518-F530